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Patients with asymmetric SNHL maybe followed with serial audiograms (every 6 months)if they have known significant asymmetric noise expo-sure or have no recent changes in hearing or wordrecognition, no risk factors for syphilis (or a negativefluorescent treponemal antibody test), and no associ-ated symptoms (such as vertigo) (Urben et al., 1999).Patients without a history of signifi cant asymmetricnoise exposure or other known cause of asymmetricSNHL, as well as those with progression of asymmet-ric SNHL, should undergo brainstem auditory-evokedresponse (BAER) testing (Urben et al., 1999). No warranty may becreated or extended by any promotional statements for this work

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duck) and level of specificity, with dispro-portionate difficulty on tasks probing subordinate ratherthan basic category or superordinate knowledge (Adlamet al., 2006; Hodges and Patterson, 2007).

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Identi? cationof the ductus is also important because as longas this is widely patent, it allows the right ven-tricle to decompress and prevents right heartfailure when the pressure becomes suprasys-temic. A cut, hemisected fresh kidney reveals two distinct regions: a cortex, the reddish-brown outer region, and a medulla, a muchlighter inner part continuous withetherrenal pelvis. The same authors concluded that thereis sufficient evidence to support a Level B (Insufficient evidence to support or refuteefficacy/safety, but established as probably effective) recommendation for use of OBTAfor the treatment of CDHA and TTH. Practical approaches include consideration of clinical CT and clinical DWImismatches in which the severity of the neurologic deficit is compared with the extent andlocation of the lesion on imaging (13–15)

Practical approaches include consideration of clinical CT and clinical DWImismatches in which the severity of the neurologic deficit is compared with the extent andlocation of the lesion on imaging (13–15). Bluearrows indicate stimulatory actionon the system; red arrows indicate inhibitory feedback. Salvioli R, Tatti M, Scarpa S, Moavero SM, Ciaffoni F, Felicetti F, Kaneski CR, Brady RO,Vaccaro AM (2005) The N370S (Asn370 > Ser) mutation affects the capacity of glucosylce-ramidase to interact with anionic phospholipid-containing membranes and saposinC

Salvioli R, Tatti M, Scarpa S, Moavero SM, Ciaffoni F, Felicetti F, Kaneski CR, Brady RO,Vaccaro AM (2005) The N370S (Asn370 > Ser) mutation affects the capacity of glucosylce-ramidase to interact with anionic phospholipid-containing membranes and saposinC. PA and lateral x-rays are obtained and fail toreveal fracture or dislocation

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